A new study reports that abnormalities in the SOD1 protein are a common factor in all types of motor neuron diseases.
A new study reports people with ALs may have a genetic mutation that causes SOD1 to aggregate motor neurons in the brain and spinal cord.
Researchers report transplanting altered neural stem cells into the brains of mice genetically engineered to exhibit symptoms of ALS, delayed the progression of the disease and extended lifespan.
A new treatment for ALS, which uses a technique that turns off the mutated SOD1 gene, has passed an early phase clinical trial for safety.
Researchers discover Alzheimer's disease, and other neurodegenerative conditions could be linked to prion-like proteins.
A new study reports an antidepressant temporarily inhibits the blood-brain barrier, allowing drugs to enter the brain. The findings could have implications for treating neurological diseases from ALS to epilepsy.
During early Alzheimer's disease, SOD1 initially weakens levels of Tau protein. However, as the disease progresses, the antioxidant becomes less effective at protecting the brain against neurodegeneration.
A new study reveals peripheral nerve damage may trigger the onset and spread of ALS in mouse models of the neurodegenerative disease.
Researchers use techniques which illuminates subtle changes in proteins and uncover new insights into the underlying causes of ALS.
Researchers report a collapse of the mitochondria-associated membrane is a common halmark in two genetic forms of ALS.
Study implicates abnormalities in SOD1 with Parkinson's disease and ALS.
Study reveals an association between intestinal inflammation and the gut microbiome in the development and progression of ALS.
