A low dose of the sedative clonazepam alleviated autistic-like behavior in mice with a mutation that causes Dravet syndrome in humans.
Scientists have observed the neurological mechanism behind temperature-dependent febrile seizures by genetically engineering fruit flies to harbor a mutation analogous to one that causes epileptic seizures in people. Their new study also highlights the first use of genetic engineering to swap a human genetic disease mutation into a directly analogous gene in a fly.
By turning skin cells of patients with Dravet syndrome into neurons, researchers create a miniature testing ground for epilepsy
Huperzine A, a compound that comes from a club moss, has been shown to increase resistance to induced seizures in mouse models of genetic epilepsy, a new study reports.
Researchers have identified a network of 320 genes they believe to be associated with epilepsy.
Researchers have identified three genes linked to hemiplegic migraine.
Researchers have identified 16 DNA regions associated with epilepsy. Eleven of the regions are newly identified.
Mice expressing a specific variant of the Scn1a gene associated with Dravet syndrome exhibited spontaneous seizures, disordered breathing and died prematurely.
Reducing levels of the Alzheimer's related Tau protein prevents autism-like behaviors in mouse models. Tau reduction also prevented seizures in the animals.
Researchers have identified a circuit within the brain that may be responsible for respiratory dysfunction and sudden death associated with Dravet syndrome.
