Mice infected with bacteria that cause mild intestinal infections exhibited Parkinson's like symptoms later in life. The findings provide a pathophysiological model in which intestinal infections may act as a trigger for Parkinson's disease.
A new study reports stress on the endoplasmic reticulum, and not mitochondrial failure, results in neurodegeneration in Parkinson's disease.
Parkinson's patients carrying mutations in PINK1 and Parkin genes have increased levels of circulating interleukin 6 and mitochondrial DNA. The findings strengthen a link between genetic risk factors from Parkinson's disease and inflammation.
Researchers identify a new chemical messenger that is critical for protecting the brain against PD.
A new study reports PINK1 triggers mitophagy, a process which breaks down and removes damaged mitochondria from cells.
Researchers report two genes associated with Parkinson's disease are also key regulators of the immune system.
According to a new study, an FDA approved drug prevented the development of Parkinson's disese in mice genetically engineered to develop the neurodegenerative disorder.