Parkinson's patients carrying mutations in PINK1 and Parkin genes have increased levels of circulating interleukin 6 and mitochondrial DNA. The findings strengthen a link between genetic risk factors from Parkinson's disease and inflammation.
Mice infected with bacteria that cause mild intestinal infections exhibited Parkinson's like symptoms later in life. The findings provide a pathophysiological model in which intestinal infections may act as a trigger for Parkinson's disease.
Researchers discover a link between neural tube defects and neurodegenerative diseases.
Researchers report two genes associated with Parkinson's disease are also key regulators of the immune system.
A new study reports stress on the endoplasmic reticulum, and not mitochondrial failure, results in neurodegeneration in Parkinson's disease.
According to a new study, an FDA approved drug prevented the development of Parkinson's disese in mice genetically engineered to develop the neurodegenerative disorder.