Constant hunger associated with Prader-Willi syndrome is, in part, the result of disordered signaling in the cerebellum, an area of the brain associated with motor control and learning.
Both physical and social factors play significant roles in depression and reports of poor wellbeing associated with obesity.
Study reveals the types of foods you load your plates with at all you can eat buffets may predict your risk for obesity.
Obese mice treated with the TSLP cytokine showed a significant loss in abdominal fat and weight. The fat loss was not associated with reduced food intake or faster metabolism, instead the cytokine stimulated the immune system to release lipids via the skin's oil-producing sebaceous glands.
Neuroimaging study reveals three distinct response types in brain areas that control hunger, food intake, and appetite in patients who had undergone weight loss surgery.
The brain regulates both eating for hunger and pleasure through serotonin-producing neurons in the midbrain, but the different types of feeding are wired by independent circuits that do not influence the other type of feeding.
Higher body fat leads to increased atrophy of the brain's gray matter and a greater risk of cognitive decline, researchers report.