Genes linked to obesity, energy metabolism, and appetite are also responsible for growth regulation in children, a new study reports.
FKBP51, a protein linked to depression and anxiety disorders, also acts as a link between the stress response system and metabolic processes.
Flavanols enhance the browning of adipose fat by activating the sympathetic nervous system. The findings could have implications for the development of new drugs to help treat obesity.
A new treatment system that focuses on the link between eating and mental health helps people with obesity make healthier decisions when it comes to eating.
Constant hunger associated with Prader-Willi syndrome is, in part, the result of disordered signaling in the cerebellum, an area of the brain associated with motor control and learning.
The functional connectivity of neural networks associated with satiety and appetite are altered prior to the development of obesity.
Both physical and social factors play significant roles in depression and reports of poor wellbeing associated with obesity.
Study reveals the types of foods you load your plates with at all you can eat buffets may predict your risk for obesity.
Obese mice treated with the TSLP cytokine showed a significant loss in abdominal fat and weight. The fat loss was not associated with reduced food intake or faster metabolism, instead the cytokine stimulated the immune system to release lipids via the skin's oil-producing sebaceous glands.
Neuroimaging study reveals three distinct response types in brain areas that control hunger, food intake, and appetite in patients who had undergone weight loss surgery.
The brain regulates both eating for hunger and pleasure through serotonin-producing neurons in the midbrain, but the different types of feeding are wired by independent circuits that do not influence the other type of feeding.
Higher body fat leads to increased atrophy of the brain's gray matter and a greater risk of cognitive decline, researchers report.
