Exposure to air pollution within the first 6 months of life alters a child's microbiome, increasing the risk for allergies, diabetes, obesity, and influencing brain development.
Children born to women with gestational diabetes and obesity were twice as likely to be diagnosed with ADHD than those born to mothers who did not suffer from those conditions.
Obesity is, in part, determined by epigenetic development in the arcuate nucleus of the hypothalamus. Findings suggest developmental epigenetics plays a significant role in both environmental and genetic influences on obesity risk.
Researchers have identified a link between metabolism and dementia-related brain measures. Obesity related to inflammation, kidney stress, or liver stress had the biggest impact on adverse brain health.
Researchers identified microRNA-7 as a non-classic risk factor for hereditary obesity.
Stress impacts the brain's response to food, researchers report. Additionally, both lean and obese people react to food cues in brain areas associated with reward and cognitive control.
High-fat diets induce hyperalgesic priming, a neurological change that represents the transition from acute to chronic pain, and allodynia or pain resulting from stimuli that do not normally provoke pain.
People who ate almonds lowered their energy intake by 300 kilojoules at their following meal. Almonds alter appetite-regulating hormones and help to reduce food intake.
Higher weight and body mass index in pre-adolescence was associated with poor brain health.
Those who report trouble sleeping are at increased risk of poor cardiometabolic health problems which can lead to type 2 diabetes.
Physically active, married females who are from financially stable backgrounds, are not obese, and do not suffer from insomnia are more likely to maintain good health and less likely to suffer cognitive, physical, or emotional problems as they age.
Mutations in the gene for the serotonin 2C receptor play a key role in obesity and dysfunctional behaviors in both human and animal models.