Stress impacts the brain's response to food, researchers report. Additionally, both lean and obese people react to food cues in brain areas associated with reward and cognitive control.
Obesity is, in part, determined by epigenetic development in the arcuate nucleus of the hypothalamus. Findings suggest developmental epigenetics plays a significant role in both environmental and genetic influences on obesity risk.
Children born to women with gestational diabetes and obesity were twice as likely to be diagnosed with ADHD than those born to mothers who did not suffer from those conditions.
Exposure to air pollution within the first 6 months of life alters a child's microbiome, increasing the risk for allergies, diabetes, obesity, and influencing brain development.
Western diets high in sugars reduce the number of Th17 inflammatory cells in the guts of mice, setting off a chain of events that lead to metabolic diseases, prediabetes, and obesity.
Researchers investigate neural pathways that meet in the brainstem which help control feeding behaviors.
Researchers have identified a molecule in the blood that is produced during exercise. The molecule, Lac-Phe, can effectively reduce food intake and obesity in mouse models.
Children with higher cardiorespiratory and muscular fitness and a lower waist-to-hip ratio had higher scores in cognitive tests for attention and processing speed during middle age.
Common epigenetic markers associated with food addiction have been identified.