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neuroinflammation

·April 29, 2025·3 min read

New Biomarker Predicts MS Progression

Researchers have identified a new biomarker in multiple sclerosis (MS): the thickness of the inflammatory rim surrounding brain lesions predicts how quickly and severely the disease progresses. By combining PET imaging and post-mortem brain analysis, the study showed that thicker rims lead to more aggressive brain damage.

Featured Neuroscience Psychology

·April 23, 2025·5 min read

Psychedelics May Reset Brain-Immune Link Driving Fear and Anxiety

A new study reveals that chronic stress activates immune cells that travel to the brain, amplify inflammation, and heighten fear responses. Researchers found that psychedelics like MDMA and psilocybin disrupt this immune-brain crosstalk, reducing stress-related fear in mice and showing similar effects in human tissue samples.

Featured Neuroscience Psychology

·April 7, 2025·8 min read

Immune Signals Shape Social Behavior and Anxiety in the Brain

New research reveals that the immune molecule IL-17 can influence behavior by acting directly on specific regions of the brain. IL-17 reduces neuron excitability in the cortex to enhance sociability, while in the amygdala, it increases excitability and induces anxiety.

This shows a brain and a bottle of herbicide.

Featured Neurology Neuroscience

·December 4, 2024·9 min read

Herbicide Exposure Linked to Long-Term Brain Inflammation, Alzheimer’s

New research reveals that glyphosate, a widely used herbicide, can cause lasting brain inflammation and accelerate Alzheimer’s-like pathology in mice. Even short-term exposure resulted in persistent inflammatory markers and neurological damage, including anxiety-like behaviors and premature death. The study found that glyphosate’s metabolite, aminomethylphosphonic acid, accumulates in brain tissue, raising concerns about its long-term safety for humans.

Featured Genetics Neurology Neuroscience

·November 29, 2024·5 min read

Spike Protein Lingers in Brain, Fuels Long COVID

A new study reveals that the SARS-CoV-2 spike protein remains in brain-protective tissues and skull bone marrow for years after infection, potentially driving long COVID's neurological symptoms. Using advanced imaging, researchers discovered elevated spike protein levels in these regions, leading to chronic brain inflammation and increased risks of neurodegenerative conditions.

Featured Neurology Neuroscience

·August 27, 2024·6 min read

COVID’s Inflammation Linked to Long-Term Brain Effects

New research uncovers how COVID-19 may cause neuroinflammation, leading to persistent neurological symptoms even after recovery. The study found elevated levels of pro-inflammatory cytokines and significant changes in cerebrospinal fluid among hospitalized patients, highlighting the brain's vulnerability to the virus.

Featured Neuroscience Psychology

·August 21, 2024·5 min read

Childhood Inflammation Linked to Later Mental Health Risks

Children with persistent inflammation are at a higher risk of developing mental health disorders like psychosis and depression in early adulthood. A study found that elevated inflammation markers, particularly around age 9, significantly increase the risk of these disorders, as well as cardiometabolic diseases such as insulin resistance. The research suggests that early-life inflammation could be a critical factor in later mental and physical health issues, prompting the need for further investigation into its role.

Featured Neurology Neuroscience

·August 19, 2024·5 min read

Long COVID Linked to Brain Inflammation and Low Cortisol

A study finds that COVID-19 proteins left in the brain may lower cortisol levels, leading to heightened inflammation and an exaggerated response to stressors. This discovery could explain the persistent neurological symptoms seen in Long COVID patients. Researchers suggest that managing stressors and investigating viral reservoirs might help alleviate these symptoms.

Featured Neurology Neuroscience

·July 12, 2024·6 min read

Infection Brain Inflammation Triggers Muscle Weakness

A new study reveals how brain inflammation from infections and neurodegenerative diseases causes muscle weakness by releasing the IL-6 protein. Researchers found that IL-6 travels from the brain to muscles, reducing their energy production and function. This discovery could lead to treatments for muscle wasting in diseases like Alzheimer's and long COVID. Blocking the IL-6 pathway may prevent muscle weakness associated with brain inflammation.

Featured Neurology Neuroscience Psychology

·April 2, 2024·5 min read

Stress and Trauma Risk Factors for Alzheimer’s

Researchers linked midlife stress and childhood trauma to an increased risk of Alzheimer’s disease and neuroinflammation. Analyzing 1,290 volunteers, researchers found that stressful life events, especially during midlife, correlate with higher levels of β-amyloid protein, crucial in Alzheimer's development, and that childhood stress is associated with later life neuroinflammation. Interestingly, the study also uncovered sex-specific effects, with stress leading to amyloid protein accumulation in men and brain atrophy in women.

Featured Neurology Neuroscience

·January 10, 2024·4 min read

Targeting Inflammation Reduces ALS Symptoms

New research reveals that in people with ALS, structural changes in neurons activate immune cells, leading to inflammation and reduced motor function. A study explores the correlation between neuron changes and immune responses.

Featured Neurology Neuroscience

·December 12, 2023·7 min read

Novel Nanoparticle Delivery System Targets Alzheimer’s Inflammation

A new study demonstrates a breakthrough in Alzheimer's disease (AD) treatment using lipid nanoparticles (LNPs) to deliver small interfering RNA (siRNA) to the brain's microglia. This approach effectively suppresses a protein linked to Alzheimer's-related inflammation.