A new drug has proven effective at restoring memories and neural connections in mouse models of Alzheimer's disease. The new drug was originally developed as a treatment for Schizophrenia. While the drug does not destroy amyloid plaques associated with Alzheimer's, it does allow the plaques to co-exist with neurons.
Researchers describe the events which cue neurogenesis in songbirds.
Researchers report bioelectrical potential is a driving force for stem cells to generate different neuron types during embryogenesis.
Researchers report axon regeneration can be boosted by using optogenetics or chemogenetics to enhance neural activity.
A new study reports participating in light physical activity, such as yoga or tai chi, can help to increase connectivity between brain areas responsible for memory formation and storage.
Researchers report exercise can promote adult hippocampal neurogenesis and improve cognition in mouse models of Alzheimer's disease. The study identifies ways to mimic the beneficial effects of experience through pharmacological and gene therapy.
BDNF released during aerobic exercise stimulates the hippocampus to sprout new neurons, a new study reports.
Severed axonal segments signal to Schwann cells to begin actin sphere formation and axon disintegration. If the process is disrupted, axon disintegration is slowed and axon fragments impair nerve regeneration.