According to a new study, the most common genetic cause of Parkinson’s is not only responsible for the condition’s distinctive movement problems but may also affect vision.
Study supports urate protection against Parkinson’s disease, hints at novel mechanism In vitro study indicates urate protection extends beyond antioxidant...
A recent study indicates microRNAs may play a far more important role in memory formation than previously thought. The research suggests microRNA—miR-182 is involved in developing memory in the amgydala.
Researchers examine mutations of the LRRK2 gene and R1441G, known as the Basque mutation, to better understand Parkinson's disease among patients in the Basque Country.
With a new insight into a model of Parkinson’s disease, researchers have identified a novel target for mitigating some of the disease’s toll on the brain.
Researchers report of elevated alpha-synuclein levels detected on the skin of patients with Parkinson's disease. The findings could offer a new biomarker for the recognition of Parkinson's before the disease reaches advanced stage.
A new finding turns one of the basics of neurobiology on its head, demonstrating that it is possible to turn one type of already differentiated neuron into another within the brain.
Researchers report that the protein Klotho plays an important role in the health of myelin, the insulating material allowing for the rapid communication between nerve cells. These findings may lead to new therapies for multiple sclerosis (MS) and Alzheimer's disease (AD).
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No one knows the cause of most cases of Alzheimer's, Parkinson's and other neurodegenerative disorders. But researchers have found that certain factors are consistently associated with these debilitating conditions. One is DNA damage by reactive oxygen species, highly destructive molecules usually formed as a byproduct of cellular respiration. Another is the presence of excessive levels of copper and iron in regions of the brain associated with the particular disorder.
Researchers discover neuronal activity can stimulate tau release from healthy neurons in the absence of cell death. The study shows treatment of neurons with known biological signaling molecules increases the release of tau into the culture medium.
Efforts to treat disorders like Lou Gehrig’s disease, Paget’s disease, inclusion body myopathy and dementia will receive a considerable boost from a new research model created by UC Irvine scientists.
Researchers visualize the molecular changes in a critical cell death protein that force cells to die. Defects in cell death have been linked to the development of diseases such as cancer and neurodegenerative conditions.