A new study reports a commonly used NSAID anti-inflammatory drug was able to reverse memory loss in mouse models of Alzheimer's disease.
A new study reports on the role the protein TDP-43 plays in the development of Alzheimer's disease.
Decreased blood flow and blood oxygen levels to the hippocampus may explain symptoms of memory loss associated with Alzheimer's. Researchers say increasing hippocampal blood flow could reverse or protect against symptoms of the neurodegenerative disorder.
A new discovery could help answer the question as to how extra ApoE4 may cause Alzheimer's disease.
A new study suggests that blood may hold clues to whether post-menopausal women may have an increased risk for brain damage that can lead to memory problems and an increased risk of stroke.
Subtle changes in thinking and memory may appear before, or in conjunction with, the development of amyloid plaques.
A new neuroprosthesis has been developed to bypass a damaged section of the hippocampus and provide a neighboring area with correctly translated memory. The device could help people with Alzheimer's disease or those who have problems with memory following brain injury.
During sepsis, the body sheds fragments of heparan sulfate, which crossed the blood-brain barrier and enters the hippocampus. The presence of heparan sulfate in the hippocampus may cause memory loss associated with septic shock.