Aging may alter neural processes for remembering continuous real-world experiences and segmenting memories.
Males with autism have atypically enhanced excitation in the medial prefrontal cortex (mPFC). This area of the brain is associated with social cognition and self-reflection. Autistic women showed a more intact mPFC response, which was associated with a better ability to camouflage social difficulties in real-world settings.
Study reveals how two neural circuits dictate the choice between social approach and avoidance. The network connecting the infralimbic cortex to the basolateral amygdala (BLA) impairs social behavior if there is a decrease in neural activity. Another network connecting the prelimbic cortex to the BLA similarly impairs social behavior if the neural activity is increased.
In the medial prefrontal cortex, loneliness is associated with a reduced representational similarity between the self and others. Feeling socially disconnected may be mirrored by a self-representation of being a "loner."
Scanning people's brains when watching videos, researchers found both neural and behavioral responses to the content could predict how long others would watch the same video on the internet.
During a conflict between two groups, oxytocin levels increase, influencing the medial prefrontal cortex. This results in a greater feeling of empathy among the group and a desire to seek revenge on rivals. The findings shed light on how conflict contagion can occur in social groups.
Researchers uncover a key role of medial prefrontal cortex corticotropin-releasing factor interneurons for bidirectionally controlling motivated behavioral styles under stress. The findings could help in the development of new treatments for PTSD.
Unique signatures of neural synchrony reflect whether pro-social or anti-social decisions are made. Brain regions, including the cingulate gyrus, amygdala, and medial prefrontal cortex, are highly synchronized when pro-social behavior is exhibited but decreases in the presence of anti-social behavior.
Methylphenidate may boost norepinephrine levels in the prefrontal cortex, which in turn regulates dopaminergic neurons firing in the striatum, when a reward is delivered. The study sheds new light on how medications for ADHD affect the reward system in the brain.