Brain folds that form during fetal development may have an impact on the age at which symptoms of frontotemporal dementia occur.
Like cognitive deficits, psychiatric symptoms such as depression, apathy, and anxiety associated with Alzheimer's disease appear to be a direct consequence of underlying brain changes as a result of increased amyloid-beta accumulation.
Researchers examine the neuroscience behind why we make certain decisions when faced with multiple choices.
The answer to what makes the human brain unique may lie in junk DNA.
Amyloid-beta accumulates in nerve cells and spreads, from cell to cell, via nerve fibers. This process occurs prior to the formation of amyloid plaques in the brain.
A new algorithm that uses data from memory tests and blood samples is able to accurately predict an individual's risk for developing Alzheimer's disease.
Researchers have identified four distinct subtypes of Alzheimer's disease. The findings could lead to more individualized treatments for those with the neurodegenerative disorder.
Study reports tau accumulation in the temporal lobe was 75% higher in women than men.
A new app combines basic data with information about specific biomarkers found in an individual's blood to assess the risk of developing Alzheimer's disease within two to four years.
Tau spreads through the human brain via neural communication pathways. The spread is accelerated by the presence of amyloid-beta.
Researchers have developed a new model of early embryonic brain development. The model, called MiSTR, allows researchers to create tissue that contains different brain regions and place them next to each other. The model produces results similar to embryonic brain development at five weeks post-fertilization.
By the age of six months, infants can recognize when an adult imitates them and perceive the imitator as more friendly.
