Researchers reveal a neurotransmitter implicated in the itch sensation plays no role in the transmission of pain sensations.
Those with eczema who produce immunoglobulin E in response to environmental allergens are more likely to experience chronic and acute itching.
Researchers have discovered a dedicated neural pathway that transmits the itchy feeling triggered by light touch.
Blocking the NPR1 receptor on the spinal cords of mice and humans may help reduce chronic itching.
Researchers have successfully blocked an immune system protein with an antibody to stop itching associated with poison ivy contact.
Researchers investigated how the nervous system interacts with the immune system to cause the inflammation and itching associated with eczema. The findings could lead to new treatment options for eczema sufferers.
According to researchers, socially contagious itching may be hard wired in the brain.
Periostin can directly activate neurons associated with itching. Blocking periostin receptors reduced itch response in mouse models of chronic allergic itching.
Researchers shed light on the mechanism that drives the uncontrollable urge to scratch an itch.
Study could help explain how pain often follows a chemical induced itch.
Researchers have discovered two distinct pathways through which a single molecule can cause both painful and itchy skin.
Researchers implicate the neuropeptide NPPB in generating the itching sensation symptomatic of eczema.
