Gene editing may provide hope for the treatment of Fragile X, the leading genetic cause of autism.
Researchers have identified a potential molecular mechanism that causes Fragile X syndrome in the developing fetal brain.
Fragile X syndrome patients with prematuration had a much earlier onset of neurological problems, including earlier symptoms of neurodegeneration. They also experienced emotional processing problems. Some of the most common emotional processing disorders reported were mood regulation, anxiety, and psychosis.
Researchers report functional alterations in L-type calcium channels were detected in models of Fragile X syndrome.
Researchers report a drug used to slow cognitive decline in Alzheimer's disease may help to reverse memory and learning problems in teens who binge drink.
Researchers report neurons in mice with a genetic defect that causes Fragile X are similar to those in mice without the syndrome, but fail to interact correctly. This failure results in the cognitive impairment associated with FXS.
A new study reports children with Fragile X syndrome show bias toward threatening rather than positive emotion.
Researchers report FMR1 and ZC3H14 proteins both regulate mRNA, which may explain their importance in memory and learning.
Researchers report a common diabetes drug helped to restore protein production in the brain to normal levels and repaired brain connections in mouse models of Fragile X.
According to researchers, maternal immune activation during pregnancy can disrupt the expression of genes and processes associated with Autism.
A new study provides additional insight into fragile X syndrome.
Two different causes of autism and intellectual disability disrupt protein synthesis at synapses, a new study reports.
