In response to injury, microglia cross the spinal boundary from the central nervous system to the peripheral nervous system. While in the PNS, microglia provide the function of clearing cellular debris at the point of injury, then return to the CNS in an altered state. Researchers propose this could account for some damage associated with neurodegenerative diseases.
A new study reports only half of infants under three months of age who have bacterial meningitis show traditional symptoms of the disease, such as high fever.
People with functional dizziness do not appear to process sensory-motor impression correctly. Instead, they rely on a stored memory model which no longer matches immediate reality.
Damage to the nasal epithelium increases the risk of bacteria entering into the brain, potentially resulting in long-term health problems.
According to researchers, as we age, it takes a lot longer to clear amyloid beta from the brain, thus leading to a build up of the toxic protein implicated in Alzheimer's disease.
At the site of injury, nerves release a protein called CXCL12 which attracts growing nerve fibers and keeps them trapped in place. This prevents the nerve fibers from growing in the correct direction to bridge the injury site.
Researchers are looking to a supercharged version of retinoic acid, which is derived from sprouts, to help develop new Alzheimer's drugs.
A tiny array of implanted electrodes may help people with spinal cord injuries to regain use of their paralyzed limbs, a new study reveals.