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beta-amyloid

·December 9, 2010·5 min read

Beta-amyloid Removal Problems May Underlie Alzheimer’s Disease

The removal of beta-amyloid is not efficient in late-onset Alzheimer's disease sufferers. These new findings could help produce better early diagnostic tests and therapies for Alzheimer's disease and related research.

Neurology

·March 6, 2012·3 min read

Scientists Pinpoint How Vitamin D May Help Clear Amyloid Plaques Found in Alzheimer’s Disease

A team of academic researchers has identified the intracellular mechanisms regulated by vitamin D3 that may help the body clear...

Featured Neurology

·March 6, 2012·2 min read

Specific Antibodies Halt Alzheimer’s Disease in Mice

Antibodies that block the process of synapse disintegration in Alzheimer’s disease have been identified, raising hopes for a treatment to...

Featured Neuroscience

·May 25, 2012·2 min read

CCR2 Involved in Removing Beta-Amyloid, Could Slow Alzheimer’s Progression

First study to suggest that the immune system may protect against Alzheimer’s changes in humans Recent work in mice suggested...

Featured Neurology

·June 13, 2012·3 min read

Researchers Identify New Group of Proteins in Brains of Alzheimer’s Patients

Researchers from Boston University School of Medicine (BUSM) have identified a novel group of proteins that accumulate in the brains...

Featured Neurology

·July 24, 2012·4 min read

New Research Determines How a Single Brain Trauma May Lead to Alzheimer’s Disease

A study, performed in mice and utilizing post-mortem samples of brains from patients with Alzheimer’s disease, found that a single event of a moderate-to-severe traumatic brain injury (TBI) can disrupt proteins that regulate an enzyme associated with Alzheimer’s. The paper, published in The Journal of Neuroscience, identifies the complex mechanisms that result in a rapid and robust post-injury elevation of the enzyme, BACE1, in the brain. These results may lead to the development of a drug treatment that targets this mechanism to slow the progression of Alzheimer’s disease.

Featured Neurology

·August 1, 2012·2 min read

Artificial Butter Flavoring Ingredient Linked to Key Alzheimer’s Disease Process

A new study raises concern about chronic exposure of workers in industry to a food flavoring ingredient used to produce the distinctive buttery flavor and aroma of microwave popcorn, margarines, snack foods, candy, baked goods, pet foods and other products. It found evidence that the ingredient, diacetyl (DA), intensifies the damaging effects of an abnormal brain protein linked to Alzheimer’s disease.

Featured Neurology

·August 9, 2012·3 min read

Blood Test for Alzheimer’s Gaining Ground

Scientists have taken a step toward developing a blood test for Alzheimer's, finding a group of markers that hold up in statistical analyses in three independent groups of patients.

Featured Neurology

·August 20, 2012·2 min read

Brain Enzyme is Double Whammy for Alzheimer’s Disease

Researchers discover that an enzyme known for generating toxic brain plaques in Alzheimer's disease also causes additional memory and cognitive deficits via a separate mechanism. They found that BACE1 disrupts the cell's production of other molecules required for PKA function. By that mechanism, BACE1 inactivates PKA and therefore inhibits memory formation in mice, even in the absence of neurotoxic β-amyloid.

The image shows a chart of the activation and antagonists of NMDA receptors.

Electrophysiology Featured Neuroscience

·February 19, 2013·2 min read

Rewriting a Receptor’s Role

Researchers upend a long-held view about the basic functioning of a key receptor molecule involved in signaling between neurons. The study describes how a compound linked to Alzheimer's disease impacts NMDA receptors and weakens synaptic connections between brain cells.

The image shows 3 diagrams of beta amyloid plaque formations.

Featured Neurology

·February 27, 2013·2 min read

Researchers Identify Possible Treatment Window for Memory Problems

A new study identifies a potential treatment window of several years for plaques in the brain considered to cause memory loss in diseases such as Alzheimer's.

This is a histopathogic image of senile plaques seen in the cerebral cortex in a patient with presenile onset of Alzheimer disease.

Featured Neuroscience

·March 5, 2013·2 min read

Green Tea Extract Interferes with the Formation of Amyloid Plaques in Alzheimers Disease

Researchers used a specific molecule in green tea extract, EGCG, to control metal associated amyloid beta aggregates associated with Alzheimer's disease and other neurodegenerative disorders.

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beta-amyloid

Beta-amyloid Removal Problems May Underlie Alzheimer’s Disease

Scientists Pinpoint How Vitamin D May Help Clear Amyloid Plaques Found in Alzheimer’s Disease

Specific Antibodies Halt Alzheimer’s Disease in Mice

CCR2 Involved in Removing Beta-Amyloid, Could Slow Alzheimer’s Progression

Researchers Identify New Group of Proteins in Brains of Alzheimer’s Patients

New Research Determines How a Single Brain Trauma May Lead to Alzheimer’s Disease

Artificial Butter Flavoring Ingredient Linked to Key Alzheimer’s Disease Process

Blood Test for Alzheimer’s Gaining Ground

Brain Enzyme is Double Whammy for Alzheimer’s Disease

Rewriting a Receptor’s Role

Researchers Identify Possible Treatment Window for Memory Problems

Green Tea Extract Interferes with the Formation of Amyloid Plaques in Alzheimers Disease

Link Between Childhood Adversity and Muscle Dysmorphia in Youth

How Imagination Fuels Empathy and Prosocial Behavior

Dopamine’s Role in Learning from Rewards and Penalties

Taste-Driven AI Algorithms Enhance Wine Selections

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