Researchers have identified a molecular mechanism that contributes to a slowed maturation of fast spiking neurons. The developmental delay has been implicated in Fragile X. However, researchers were able to restore normal maturation speed by administering an activator of the TrkB protein.
Researchers successfully used a synthetic compound to stimulate a receptor pathway to promote remyelination in the brain. The technique may have significant beneficial implications for treating multiple sclerosis, researchers say.