A new study reports people with BDD and anorexia have similar brain abnormalities which affect their ability to process visual information.
A small study suggests deep brain stimulation may be beneficial for improving mood and reducing anxiety while helping to increase weight in those with anorexia.
The brains of those with anorexia have sizable reductions in three critical measures, including cortical thickness, subcortical volume, and cortical surface area.
Eating disorder behaviors are reinforced due to changes in the brain's reward response processes and alterations in the food intake control network.
The largest delay discounting effects were found to be associated with bipolar disorder, borderline personality disorder, and schizophrenia. The image is in the public domain.
A new study reports that, even after extensive treatment and weight gain, the brains of those with anorexia may remain altered, leaving them at additional risk of relapse.
Researchers report gut bacteria could play a vital role in anorexia nervosa.
Hyperactivity in a serotonin-dopamine circuit in the brain appears to be responsible for persistent anorexia in animal models. The DRD1 receptor appears to drive the hyperactivity. Deleting the DRD1 gene restored normal eating behaviors in animals.
Genome wide analysis of anorexia identifies the common roots the disease shares with metabolic and psychiatric traits.
Researchers have revised outdated estimates of the prevalence of eating disorders in the US. The study estimates 0.80% of people will suffer from anorexia during their life time, while 0.28% will be affected by bulimia and 0.85% of people will suffer from binge eating disorders.
Children with autistic traits at age seven were 24% more likely to develop weekly eating disorders, including fasting, purging, and binge eating, by age 14.
Researchers report when food is absent, Agrp neurons initiate repetitive behaviors.
