A new study reveals sex differences in the way those with alcohol use disorder process facial emotions. Men with AUD showed greater activation in frontal brain areas when processing facial emotions. The increased activation was not seen in women with AUD.
Men with alcohol use disorder have diminished brain activity in areas associated with emotional processing, memory and social processing, compared to women with AUD. The findings may lead to gender-specific treatments to help relieve addition to alcohol.
Genome-wide study identifies five novel alcohol use risk loci which can pass on the risk of developing alcohol abuse disorder from parents to children.
Neural patterns of activity in the medial prefrontal cortex associated with the intention to drink alcohol are influenced by the genetic risk for alcohol use disorder.
People who are most sensitive to the pleasurable and rewarding effects of alcohol are at greater risk of developing alcohol use disorders.
Ketamine reduced alcohol intake in male rat models of alcohol use disorder but increased the desire for alcohol in low-consumption females.
Drinking small amounts of alcohol, like one pint of beer or a large glass of wine, significantly impairs our feeling of being in control of our actions. A new study reveals even one beer can lead to overconfidence in driving ability and make us act in inappropriate, potentially dangerous ways. The study begs the question, are current alcohol limits for driving truly safe?
Twenty-nine genes have now been identified as being linked to problematic alcohol use. A new study report, in addition to an increased risk of alcohol use disorder, people with specific genes linked to AUD also have an increased risk of depression, insomnia, and addiction to tobacco.
A single shot of ketamine administered to heavy drinkers following reactivation of their drinking memories led to a rapid decrease in the urge to drink. The effect lasted for over nine months.
Administering oxytocin blocks the enhanced motivation for drinking alcohol that fuels alcohol use disorder by blocking GABA signaling in the central nucleus of the amygdala.