A new series of studies review the roles neuroplasticity and neurogenesis play in alcohol addiction and recovery.
People who are most sensitive to the pleasurable and rewarding effects of alcohol are at greater risk of developing alcohol use disorders.
Manipulating memory via optogenetics mitigated addiction-related behaviors.
Study identifies inflammatory mechanisms and cellular activity in the amygdala that drives alcohol addiction in mice. Chronic alcohol exposure compromises immune cells in the brain, driving anxiety and alcohol consumption that may lead to the development of AUD.
Male and female rodents exhibit distinct symptoms and brain features of both PTSD and alcohol use disorder. Following trauma, males show increased GABA receptor function, while females showed increased GABA release.
Having a parent with an alcohol use disorder affects how your brain transitions between active and resting states, regardless of your own drinking habits.
Optogenetic inactivation of CRF neurons in the central nucleus of the amygdala decreases escalation of alcohol consumption and intensity of withdrawal in rodent models of alcoholism. The findings suggest a potential target for treating excessive drinking in alcohol use disorder.
A new study reveals the combination of ketamine with naltrexone can help treat symptoms of both depression and addiction.
According to researchers, those who live in colder regions with less daytime sun light drink more alcohol than those who live in warm areas. Climate, researchers say, may impact the prevalence of alcoholism and alcoholic cirrhosis.
Activating a receptor found in neurons in the habenula could help treat alcohol addiction and ease withdrawal symptoms, researchers report.
According to a new Frontiers in Behavioral Neuroscience study, children who experienced TBI under the age of 5 were 3.6 times more likely to have problems with substance abuse as teens. Finding suggest traumatic brain injuries during early life could be a risk factor for alcohol abuse later in life.