Researchers found a key difference in the brains of people with Alzheimer’s disease and those who are cognitively normal but still have brain plaques that characterize this type of dementia.
No one knows the cause of most cases of Alzheimer’s, Parkinson’s and other neurodegenerative disorders. But researchers have found that certain factors are consistently associated with these debilitating conditions. One is DNA damage by reactive oxygen species, highly destructive molecules usually formed as a byproduct of cellular respiration. Another is the presence of excessive levels of copper and iron in regions of the brain associated with the particular disorder.
Indications of Alzheimer’s disease may be evident decades before first signs of cognitive impairment
Researchers at Mount Sinai School of Medicine have found that patients with Alzheimer’s disease have lower glucose utilization in the brain than those with normal cognitive function, and that those decreased levels may be detectable approximately 20 years prior to the first symptoms of Alzheimer’s disease.
A new study implicates microRNAs as a possible molecular switch which could contribute to a risk factor for panic disorder.
A new study suggests, on average, people with greater hippocampal volume are better able to recover from post traumatic stress disorder than those with reduced hippocampal volume.
The claim that stress can shorten your life isn’t necessarily true, according to researchers at UC Riverside. Undertaking a twenty year long study into personality as a predictor of longevity, researchers have discovered that many of the conventional pearls of wisdom we hear about extending personal lifespan are incorrect. Researchers discovered the key to a […]
Neuroscientists have provided an insight into the neuronal mechanisms involved in prosocial behavior, and how they are modulated by perceived group membership. Researchers suggest that there is a neurobiological basis of whether people chose to help, or withdraw help, based on positive or negative identification of the person in pain.
Neuroscience research published in September’s Journal of Neuroscience suggests early life stresses may modify the GAD1 gene, which controls the production of GABA. Through their research on rats, researchers were able to note that those who experienced a lack of affection showed an obstruction within the DNA which controls the GAD1 gene. As it is believed that GABA deficits might be apparent within schizophrenic patients, researchers propose that the modification of GAD1 might determine a child’s predisposition to mental illness.
Neuroscientists at USC have discovered that carbonated drinks set off the same pain sensors in the nasal cavity as mustard, albeit at a lower intensity. During experimentation, researchers flowed carbonated saline over a dish containing nerve cells taken from the sensory circuits in the nose and mouth. They discovered that the gas activated a specific cell which serve as general pain sensors and expresses the TRPA1 gene.